朱卫忠：男，1964年11月生，特聘教授，药理学与内科学研究生导师。1998年博士研究生毕业于北京医科大学病理 生理专业，获医学博士学位（师从著名学者韩启德教授）；1999年获首届全国优秀博士论文一等奖，1999-2010：美国NIH博士后、助理研究员、研 究员；美国Temple大学医学院助理教授。从事心血管疾病（分子心脏与信号传导）基础研究20多年，特别是近年来首次发现CaMKII参与肾上腺素受体 致细胞凋亡的信号传导、炎症反应及心肌缺血损伤机制，并相继以第一作者或通讯作者在J Clin Invest、Circ Res 、Proc Natl Acad Sci、J Biol Chem等SCI高影响因子国际权威杂志发表研究论文30多篇，在分子心血管病研究领域具有较高的学术造诣。
通讯作者（或第一作者）代表作：
1．G(i)-Biased beta(2)AR Signaling Links GRK2 Upregulation to Heart Failure CIRCULATION RESEARCH,2012JAN,110(2):265-73
2．Activation of CaMKII-delta C is a common intermediate of diverse death stimuli-induced heart muscle cell apoptosis． J Biol Chem． 2007, 282(14):10833-9
3．Cheng Ca2+/calmodulin kinase II-dependent phosphorylation of ryanodine receptors suppresses Ca2+ sparks and Ca2+ waves in cardiac myocytes．Circulation research, 2007 100(3):399-407．
4． The Enigma of ß2-Adrenergic Receptor Gi Signaling in the Heart The Good, the Bad, and the Ugly． Circulation research, 2005, 97;507-509
5．2-Adrenergic Receptorb1- and bDimerization of -Adrenergic Modulation of Cardiac Contractility． CirbSubtypes Optimizes Res, 2005, 97:244-251
6．Emerging Concepts and Therapeutic Implications of ß -Adrenergic Receptor Subtype Signaling． Pharmacology & Therapeutics, 2005;108(3):257-68
7．1-Adrenergic Stimulation ModulatesbSustained Cardiac Contractility by Ca2+/Calmodulin Kinase Signaling Pathway． Circ Res． 2004;95(8):798-806．
8．-adrenoceptor signaling pathwaysbSubtype-specific in the heart and their potential clinical Implications, TIPS 2004, 25(7):358-65．
9．1-Adrenergic Stimulation to ApoptoticbLinkage of Heart Cell Death Through PKA-Independent Activation of Ca2+/Calmodulin Kinase II．J Clin Invest． 2003 , 111(5):617-25．
10．Dual modulation of cell survival and cell death by beta 2-adrenergic signaling in adult mouse cardiac myocytes． Proc Natl Acad Sci U S A． 2001; 98:1607-1612．